Israel Coronel (@icoronelm) header
Israel Coronel (@icoronelm) profile picture

Israel Coronel

@icoronelm

Indiana University School of Medicine

Joined February 2022
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  • Followers56
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Photo by Israel Coronel (@icoronelm)
I am so excited to share that I was recently awarded the Alzheimer’s Association Mel Perelman Fellowship for Alzheimer’s Research. This is such an honor! Thank you to the Perelman donors, Dr. Landreth, @miguelcmoutinho, @alzassociation, #ALZFunded, @NeurosciStark, @IUMedSchool.
Image from Israel Coronel (@icoronelm)
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I am thrilled to announce that I was awarded the 2024 Alzheimer’s Association Research Fellowship! Thank you to Dr. Landreth, Dr. Moutinho @miguelcmoutinho, Landreth’s Lab, Alzheimer’s Association @alzassociation, #ALZFunded and at @NeurosciStark @IUMedSchool for their support!
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Gary Landreth, PhD, @miguelcmoutinho and Jared Brosch, MD at @IUMedSchool collaborated to translate a basic science finding into a clinical trial for Alzheimer's disease. #goteamstark #translationalresearch medicine.iu.edu/blogs/neuros…
Alzheimer's disease basic science finding leads to clinical trial

Alzheimer's disease basic science finding leads to clinical trial

A novel therapeutic target to treat Alzheimer’s disease identified in 2022 by Indiana University School of Medicine researchers will now be translated to a clinical trial, spearheaded through a...

medicine.iu.edu
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Excited to share our new study about how TREM2 splice isoforms can contribute to a pool of heterogenous sTREM2 and affect synaptic plasticity. @Mad4Microglia @Andy_P_Tsai @icoronelm @LasagnaLab @BradyAtwood @pbmartinc @KwangsikN @btlamb genomemedicine.biomedcentral…
TREM2 splice isoforms generate soluble TREM2 species that disrupt long-term potentiation

TREM2 splice isoforms generate soluble TREM2 species that disrupt long-term potentiation

Genome Medicine - TREM2 is a transmembrane receptor expressed by myeloid cells and acts to regulate their immune response. TREM2 governs the response of microglia to amyloid and tau pathologies in...

link.springer.com
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Congratulations to the Landreth Lab @miguelcmoutinho at SNRI for this exciting new study on how niacin limits Alzheimer's disease progression in animal models. medicine.iu.edu/news/2022/03…
Researchers discover intake of FDA-approved drug modulates disease progression in Alzheimer’s...

Researchers discover intake of FDA-approved drug modulates disease progression in Alzheimer’s...

Indiana University School of Medicine researchers found that niacin limits Alzheimer’s disease progression when used in models in the lab, a discovery that could potentially pave the way toward...

medicine.iu.edu
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Congratulations!! @miguelcmoutinho
Miguel Moutinho (@miguelcmoutinho) profile pictureMiguel Moutinho@miguelcmoutinho23 Mar 2022
Our paper about the role of the niacin receptor HCAR2 in AD was just published! Congratulations and thank you for all the help and support. Let's celebrate! @Andy_P_Tsai @pbmartin86 @LasagnaLab @Peter_BC_Lin @adrianoblak @btlamb @icoronelm @NeurosciStark science.org/doi/10.1126/scit…
The niacin receptor HCAR2 modulates microglial response and limits disease progression in a mouse...

The niacin receptor HCAR2 modulates microglial response and limits disease progression in a mouse...

The niacin receptor (HCAR2) modulates the microglia response to amyloid pathology and affects disease progression in a mouse model of Alzheimer’s disease.

science.org
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Our paper about the role of the niacin receptor HCAR2 in AD was just published! Congratulations and thank you for all the help and support. Let's celebrate! @Andy_P_Tsai @pbmartin86 @LasagnaLab @Peter_BC_Lin @adrianoblak @btlamb @icoronelm @NeurosciStark science.org/doi/10.1126/scit…
The niacin receptor HCAR2 modulates microglial response and limits disease progression in a mouse...

The niacin receptor HCAR2 modulates microglial response and limits disease progression in a mouse...

The niacin receptor (HCAR2) modulates the microglia response to amyloid pathology and affects disease progression in a mouse model of Alzheimer’s disease.

science.org
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