BsN| RN๐ฉบ | ุตูุจูุฑ | ๐ธ Cervical Cancer Screening Advocate๐ธ | ๐ฐ๐ช.๐น๐ฟ | Resin Crafter IG: craftedbysevika
Nairobi, Kenya
Joined February 2020
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A baby goat started pushing a cat, but the cat thought it was just getting cuddles
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You can learn ECG 10 times and still be confused when you're reading one.
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Man, I really didn't need to know my governor is a real eater ๐ญ๐ญ๐ญ
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Answer: B. Hypokalemia
Potassium is essential for maintaining the resting membrane potential of skeletal muscle cells. Low serum potassium can disrupt neuromuscular function, causing muscle weakness, cramps, and, in severe cases, paralysis or dangerous cardiac arrhythmias.
Clinical pearl: Muscle cramps are not specific to hypokalemia. Hypocalcemia and hypomagnesemia can also cause muscle cramps or neuromuscular hyperexcitability.
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why is life so draining i just want money & head
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One would think you're objectively attractive tweeting this ๐๐๐คทโโ๏ธ
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Magnesium sulfate (IV) is the drug of choice for torsades de pointes, even if the serum magnesium level is normal. If the patient is unstable or pulseless, immediate unsynchronized defibrillation is required.
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If my pussy can grow hair thicker than your beards, I suggest you lower your tone when you talk to me
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Due to inflation I'm downgrading from ADHD to AD720p with ads
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The gym isnโt hard.
The eating is.
The fucking eating.
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โ a. The drug of choice for acute gout attack is Colchicine.
โข Colchicine is one of the 1st-line treatments in acute gout for rapid relief of inflammation & pain, ideally started within 24 hours of attack onset.
โข Other 1st-line options include NSAIDs (e.g., indomethacin) & corticosteroids (oral or intra-articular) especially in patients contraindicated for NSAIDs.
โข Allopurinol & febuxostat are urate lowering therapies, used long term for prevention of gout attacks but should not be started or stopped during an acute attack.
โข Probenecid is a uricosuric agent used for chronic gout management Not for acute attacks.
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Anticoagulants are used for venous thrombosis because venous clots are rich in fibrin formed by slow blood flow (stasis) and the coagulation cascade, whereas antiplatelets are used for arterial thrombosis because arterial clots are driven by platelet aggregation triggered by high-shear stress and ruptured atherosclerotic plaques.
