cityOwl

Going to chime in here, but the Mitochondria don't "shift into survival" - they straight up 'kick it'. Especially in #LongCovid, causing things like: fatigue, brain fog, and inflammation. Stating adoption of some 'transitional state' such as a "Shift" is not correct. -In acute COVID-19, SARS-CoV-2 directly 'impairs' mitochondria by disrupting ATP production, increasing reactive oxygen species (ROS), and damaging mitochondrial structure (e.g., swollen cristae). -In Long COVID, persistent mitochondrial 'dysfunction' evidenced by suppressed oxidative phosphorylation, reduced fatty acid oxidation, and elevated lactate - contributes to symptoms, alongside inflammation, neuroinflammation, and endothelial dysfunction. Now apply that to kids as they grow and get repeatedly exposed. Their mitochondria don't go to sleep, they are damaged. -Studies (e.g., muscle biopsies, CPET) show clear mitochondrial failure, not a "survival mode." -Your post oversimplifies a multifactorial condition, ignores viral and immune-mediated damage, and lacks evidence for its treatment implications. Long COVID symptoms arise from complex mechanisms, requiring multidisciplinary care, not just nutritional fixes. References: DOI:10.3390/ijms24032050, DOI:10.1038/s41598-024-66873-3.